Diabetes as a consequence of COVID-19: A case report from Na
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The SARS-CoV-2 coronavirus, which causes the coronavirus disease COVID-19, can penetrate many different body cells. It can also penetrate the so-called beta cells in the pancreas and damage them, as a research team involving the Cluster of Excellence "Precision Medicine in Chronic Inflammation" has now observed for the first time.

The publication is an initial description of insulin deficiency diabetes after a COVID-19 illness, based on an observed case. "A 19-year-old patient came to us in the clinic with newly-developed severe diabetes with insulin deficiency. It could be shown that he apparently had experienced a SARS-CoV-2 infection a few weeks before," reported researchers.

"Such an insulin deficiency diabetes, i.e. type 1 diabetes, is usually triggered by an autoimmune response, in which the immune system incorrectly identifies the beta cells in the pancreas as foreign and attacks them. But this autoimmune response was not present in this patient. We assume that here, the SARS-CoV-2 virus itself attacked the beta cells," said researchers.

This also fits with the fact that the beta cells possess a crucial receptor: the ACE2 receptor. The SARS-CoV-2 virus can specifically bind with this receptor. The receptor is also used by the virus as an entry point into the other body cells which it attacks.

Source: https://www.nature.com/articles/s42255-020-00281-8
Dr. T●●●●z H●●●●●●i and 7 others like this5 shares
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D●●a M●●e
D●●a M●●e General Medicine
Does this mean that people with diabetes gets more sever illness due to the neta cell distruction which is already present?
Sep 3, 2020Like