Patent ductus venosus and CHD: a case report with review
Failure of the ductus venosus to close may result in galactosemia, hypoxemia, encephalopathy with hyperammonia, and hepatic dysfunction.

Published in the journal Cardiology Research, the authors report an infant with complex congenital heart disease (CHD) who developed coagulopathy and hyperammonia during the preoperative period secondary to patent ductus venosus (PDV). Previous reports of PDV in CHD are presented, its etiology and clinical consequences reviewed, and options for therapeutic treatment discussed.

Once the diagnosis was made, hybrid stage 1 palliation was performed to allow for discontinuation of the prostaglandin infusion and facilitate PDV closure.

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